a few days of dark before harvest???

Brick Top said:

The Stichting Institute of medical Marijuana (SIMM), the first company to sell marijuana through the pharmicies of Holland, has been investigating the medical possibilities of cannabis, together with TNO laboritories and the University of Leiden.

One of their discoveries has been that to keep the ripe plants in the dark before harvesting could increase their potency.

SIMM's growers seperated a crop of mature plants, harvested half of them and kept the other half in absolute darkness for 72 hours before cutting and drying.

Analysis of the resulting dried buds showed that some varieties had seen increases of THC of up to 30 %, while the CBD and CBN remained the same.

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businessmen said:

Do you guys see a difference on the magnifying glass after the dark? What is actually happening? More new clear trichomes pop up? Maybe you just really like the high of clear trichomes and should really harvest sooner, not necessarily put them in 3 days dark. Just throwing out ideas.

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socomplicated said:

i asked a question smart ass..... y would you put a plant in 12 hours of darkness?? where is the line crossed on hours of darkness at the end of flowering? and scientificly explain it... if you can come up with a better explanation than bricktop of y it doesnt help, then i might believe you... but youre doin the same thing by sayin its bullshit and doesnt work, when its abviously been tested... honestly, all of us noobs get our fuckin "retarted noob" information from retarded fuckin "know it all assholes" like yourself.... if someone told me that your mom sucked dick better after 24 hours in the dark, you better believe im gonna put that bitch in pure darkness for 24 hours....

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rabidcow said:

here is the strain description from my widow from atitude flying dutchman seeds. Copied and pasted.

It's suggested to flower (12 hour light) this marijuana variety for 8 weeks, but 10 weeks will really give her the crystals you are after. Try to turn the lights off altogether the final 2 weeks of the flowering cycle- or at least down to 8 hours. This keeps the flowers from re-growth and stresses the plant into giving up its last drop of goodness as crystal to protect the flower! The buds have so much THC on them that it is hard to see them at all.

Ok, same horse is dead lets beat it from this point of view. Why the fuck is flying dutchman going to tell you to " Try to turn the lights off altogether the final 2 weeks "?
I love the phrase "Try to" here. What the fuck do they mean try to?
Either do it or dont.
But the idea that this seed distributor would put/pay a guy or girl in a desk, To write that up if they didnt feel it would benifit the consumer makes no sense. I mean, we have all read some shit in these strain descriptions that just kinda made ya laugh cause it is basicly the same shit just a little processed through the thesaurus. I even dispute some of the claims some sites/seed banks make as to what it could cure/help. Just find this particular description fucking out there, I bought it tho i guess lol.
So, they say last 2 weeks...... no light........ I would pay 50 bucks to have a conversation with anybody who actualy did this. This isnt an offer, i mean on what planet could i make this offer and you wouldnt have a 99% rate of fucking liars looking for a free 50 bucks
Ok i am high you caught me. on topic. Lights off for 2 weeks and let my plants just sit there. If i had 100 plants in pots, sure i would put 1 in the dark for 2 weeks. But I dont.
Still tho, why Flying Dutchmen White Widow Feminized peoples say that?

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Smokealotapotamus said:

Is it possible that instead of stressing them into producing more resin instead they produce male flowers as their last resort? And if so, is it possible that when you cut and hang the buds they could begin seed production since they are still growing to some degree? Or is it pretty much risk free to leave them in the dark?

I am curious cause my plants are about to 12 hours of darkness and ready to harvest..fighting the urge to do it tonight instead of in another day and a half.

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Demosthenese said:

ill go brush up on my mj anatomy/chemical production before i start throwing technical terms like THC around, fair enough, but it is a fact that you can get honey oil from the stems and leaves of a vegging plant because i do it ALL THE TIME lol, and im pretty sure minute amounts of THC are present in all parts of the plant, including the seeds.
I've done it to bud too, and while the quality is probably better, it's not signifigant. Hypothetically try smoking some stem; it will get you high, if only a little and with a mad headache. If you pack some stem in a pipe and then only smoke the outside layer off of them it will be less horrible, but butane extraction of molecules of THC is definetly the way to go.
So I'll admit a lack of knowledge of what chemical (THC, CBC, CBN as the main canibinoids) it is that is still getting me high since there are hundreads of canabinoids and they all contribute to the high. But you can definetly do it; i just don't want nubs to start throwing away stem they could be making into free hash

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Neurotoxic Effects from Butane Gas

Acute Intoxication
Because butane gas inhalants enter through the pulmonary system, they immediately enter into the blood supply and within seconds produce intoxication. The acute effects of inhalants include dizziness, hypertension (increased blood pressure), tachycardia (increased heart rate), impaired coordination, disorientation, temporal distortion, confusion, thick slurred speech, delirium, hallucinations, assaults and suicide attempts. Depending upon the inhalant, recovery may take minutes to hours or may not occur at all. Single episode use can be fatal because of oxygen displacement from red blood cells, hypoxia and asphyxiation. Victims of pulmonary effects are often found with a paper bag over the head.

Profound relaxation and deep sleep usually follow the initial euphoric phase.Unpleasant symptoms reported after the use of inhalants include agitation, seizures, ataxia, headache, and dizziness.

Chronic Effects
Chronic inhalant abuse destroys motor neurons that send commands from the brain to the hands and feet. As these motor neurons fail, varying degrees of motor impairment result, including a decreased ability to perform manual and mental tasks. For example, toluene vapors produce high levels of this lipid soluble chemical, particularly in the brain. Toluene abusers present symptoms of motor uncoordination, fatigue, mental impairment, and increasingly greater degrees of permanent central nervous system damage. Most inhalants produce some degree of hepatotoxicity (liver damage). Halogenated hydrocarbons, such as freon, cause severe hepatotoxicity.

Some inhalants change cardiac physiology and increase the risk for cardiac failure. For example,butane (from cigarette lighters), freon (from aerosol propellants) and toluene (from glues) hypersensitize cardiac cells to norepinephrine, the neurotransmitter that stimulates cardiac contractions. Inhalants interfere with the transport of oxygen by interfering with the binding or release of oxygen by red blood cells. The resulting hypoxia also causes cardiac cell hypersensitivity to norepinephrine. Norepinephrine sensitivity and hypoxia can cause cardiac muscles to defibrillate or begin contracting randomly. A syndrome called Sudden Sniffing Death (SSD) occurs without warning, and discontinuation of breathing the inhalant does not reverse the sequence of events. Victims of SSD often appear to sense that something is wrong, and run away from the source or site where they were inhaling, before collapsing and dying.

Neurotoxic Effects
Permanent cerebral and cerebellar neurological disability is the most well known toxic effect of chronic inhalant abuse. Long-term abusers are at significant risk for a neurological syndrome consisting of memory loss, cognitive impairment, sleep disturbance, depression, anxiety, and personality changes. Permanent cognitive disorders are also well described in patients who chronically sniff gasoline. Long term occupational chemical exposure (e.g., painters) may result in the development of cerebral atrophy and abnormal EEGs.

Chronic abuse of n-hexane and nitrous oxide are well known to cause peripheral neurological deficits including profound sensorimotor polyneuropathy (n-hexane) and a demyelinating polyneuropathy and extremity weakness (nitrous oxide), which appears to be related to the inactivation of vitamin B12, an important cofactor in many necessary biochemical reactions.

Inhalation of leaded gasoline increases the risk for neurological complications from organic lead poisoning. These include mental confusion, poor short-term memory, psychosis, and encephalopathy. Symptoms of inorganic lead poisoning (headache, abdominal pain, hepatic injury, renal damage) have also been reported in patients who chronically inhale gasoline.

Animal and human research shows that most inhalants are extremely toxic. Perhaps the most
significant toxic effect of chronic exposure to inhalants is widespread and long-lasting damage to
the brain and other parts of the nervous system. For example, both animal research and human
pathological studies indicate that chronic abuse of volatile solvents such as toluene damages the
protective sheath around certain nerve fibers in the brain and peripheral nervous system. This
extensive destruction of nerve fibers is clinically similar to that seen with neurological diseases
such as multiple sclerosis.

The neurotoxic effects of prolonged inhalant abuse include neurological syndromes that reflect
damage to parts of the brain involved in controlling cognition, movement, vision, and hearing.
Cognitive abnormalities can range from mild impairment to severe dementia. Other effects can
include difficulty coordinating movement, spasticity, and loss of feeling, hearing, and vision.
Inhalants also are highly toxic to other organs. Chronic exposure can produce significant damage
to the heart, lungs, liver, and kidneys. Although some inhalant-induced damage to the nervous
and other organ systems may be at least partially reversible when inhalant abuse is stopped,
many syndromes caused by repeated or prolonged abuse are irreversible.
Abuse of inhalants during pregnancy also may place infants and children at increased risk of
developmental harm. Animal studies designed to simulate human patterns of inhalant abuse
suggest that prenatal exposure to toluene or trichlorethylene (TCE) can result in reduced birth
weights, occasional skeletal abnormalities, and delayed neurobehavioral development. A number
of case reports note abnormalities in newborns of mothers who chronically abuse solvents, and
there is evidence of subsequent developmental impairment in some of these children. However,
no well-controlled, prospective study of the effects of prenatal exposure to inhalants in humans
has been conducted, and it is not possible to link prenatal exposure to a specific chemical to a
specific birth defect or developmental problem.

References
Brick, J. (199. Inhalants, Technical Document No. 3. Yardley, PA: Intoxikon International.

Broussard, L. (1999). Inhalants. In B. Levine (Ed.). Principles of forensic toxicology (pp 345-353). Washington: American Association for Clinical Chemistry.

Kolecki, P and Shih, R. (2003). Inhalant abuse. In J. Brick (Ed.). Handbook of the medical consequences of alcohol and drug abuse (pp 579-607). New York: Haworth Medical Press.

BDOGKush said:

Waste of time

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