Phosphorus (P)
Phosphorus is a component of certain enzymes and proteins, adenosine triphosphate (ATP), ribonucleic acids (RNA), deoxyribonucleic acids (DNA) and phytin. ATP is involved in various energy transfer reactions, and RNA and DNA are components of genetic information.
Phosphorus (P) deficiency:
Figure 11 is severe phosphorus (P) deficiency during flowering. Fan leaves are dark green or red/purple, and may turn yellow. Leaves may curl under, go brown and die. Small-formed buds are another main symptom.
Phosphorus deficiencies exhibit slow growing, weak and stunted plants with dark green or purple pigmentation in older leaves and stems.
Some deficiency during flowering is normal, but too much shouldn't be tolerated. Red petioles and stems are a normal, genetic characteristic for many varieties, plus it can also be a co-symptom of N, K, and Mg-deficiencies, so red stems are not a foolproof sign of P-deficiency. Too much P can lead to iron deficiency.
Purpling: accumulation of anthocyanin pigments; causes an overall dark green color with a purple, red, or blue tint, and is the common sign of phosphate deficiency. Some plant species and varieties respond to phosphate deficiency by yellowing instead of purpling. Purpling is natural to some healthy ornamentals.
Figure 11
Figure 12 shows Phosphorus (P) deficiency during vegatative growth. Many people mistaken this for a fungus, but look for the damage to occur near the end of leave, and leaves the color dull greyish with a very brittle texture.
Figure 12
Phosphorus (P) Toxicity:
This condition is rare and usually buffered by pH limitations. Excess phosphorus can interfere with the availability and stability of copper and zinc.
Potassium (K)
Potassium is involved in maintaining the water status of the plant and the
tugor pressure of it's cells and the opening and closing of the stomata. Potassium is required in the accumulation and translocation of carbohydrates. Lack of potassium will reduce yield and quality.
Potassium deficiency:
Older leaves are initially chlorotic but soon develop dark necrotic lesions
(dead tissue). First apparent on the tips and margins of the leaves. Stem and branches may become weak and easily broken, the plant may also stretch. The plant will become susceptible to disease and toxicity. In addition to appearing to look like iron deficiency, the tips of the leaves curl and the edges burn and die.
Potassium - Too much sodium (Na) displaces K, causing a K deficiency. Sources of high salinity are: baking soda (sodium bicarbonate "pH-up"), too much manure, and the use of water-softening filters (which should not be used). If the problem is Na, flush the soil. K can get locked up from too much Ca or ammonium nitrogen, and possibly cold weather.
Figure 13
Figure 14
Potassium (K) Toxicity:
Usually not absorbed excessively by plants. Excess potassium can aggravate the uptake of magnesium, manganese, zinc and iron and effect the availability of calcium.
Magnesium (Mg)
Magnesium is a component of the chlorophyll molecule and serves as a cofactor in most enzymes.
Magnesium (Mg) deficiency:
Magnesium deficiency will exhibit a yellowing (which may turn brown) and interveinal chlorosis beginning in the older leaves. The older leaves will be the first to develop interveinal chlorosis. Starting at leaf margin or tip and progressing inward between the veins. Notice how the veins remain somewhat green though as can be seen in figure 15.
Notice how in figure 16 and 17 the leaves curl upwards like they're praying? They're praying for Mg! The tips may also twist.
This can be quickly resolved by watering with 1 tablespoon Epsom salts/gallon of water. Until you can correct nutrient lockout, try foliar feeding. That way the plants get all the nitrogen and Mg they need. The plants can be foliar feed at ½ teaspoon/quart of Epsom salts (first powdered and dissolved in some hot water). When mixing up soil, use 2 teaspoon dolomite lime per gallon of soil.
If the starting water is above 200 ppm, that is pretty hard water, that will lock out mg with all of the calcium in the water. Either add a 1/4 teaspoon per gallon of epsom salts or lime (both will effectively reduce the lockout or invest into a reverse osmosis water filter.
Mg can get locked-up by too much Ca, Cl or ammonium nitrogen. Don't overdo Mg or you'll lock up other nutrients.
Figure 15
Figure 16
Figure 17
Magnesium (Mg) Toxicity:
Magnesium toxicity is rare and not generally exhibited visibly. Extreme high levels will antagonize other ions in the nutrient solution.
Zinc (Zn)
Zinc plays a roll in the same enzyme functions as manganese and magnesium. More than eighty enzymes contain tightly bound zinc essential for their function. Zinc participates in chlorophyll formation and helps prevent chlorophyll destruction. Carbonic anhydrate has been found to be specifically activated by zinc.
Zinc Deficiencies:
Deficiencies appear as chlorosis in the inter-veinal areas of new leaves producing a banding appearance as seen in figure 18. This may be accompany reduction of leaf size and a shortening between internodes. Leaf margins are often distorted or wrinkled. Branch terminals of fruit will die back in severe cases.
Also gets locked out due to high pH. Zn, Fe, and Mn deficiencies often occur together, and are usually from a high pH. Don't overdo the micro-nutrients, lower the pH if that's the problem so the nutrients become available. Foliar feed if the plant looks real bad. Use chelated zinc. Zinc deficiency produces "little leaf" in many species, especially woody ones; the younger leaves are distinctly smaller than normal. Zinc defeciency may also produce "rosetting"; the stem fails to elongate behind the growing tip, so that the terminal leaves become tightly bunched.
Figure 18
Zinc Toxicity:
Excess Zinc is extremely toxic and will cause rapid death. Excess zinc interferes with iron causing chlorosis from iron deficiency. Excess will cause sensitive plants to become chlorotic.
IMMOBILE ELEMENTS
Immobile elements will show their first symptoms on younger leaves and progress to the whole plant.